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Brain damage caused by prenatal alcohol exposure

Abraham Rosenberg
From: Science & Medicine: Volume 3 Number 4: Page 42 (August 1996)

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Abstract: Embryonic development of brain neurons depends upon biosynthesis of gangliosides that govern neuronal development mechanisms. When the developing neuron is exposed to moderate levels of alcohol, key turning points that regulate the biosynthesis of required species of gangliosides are blocked, embryonic brain developmint is aberrant, and the CNS component of fetal alcohol syndrome is and expected outcome. The alcohol-inhibited events are phophorylation of microtubule-associated proteins and endocytosis of neurotrophic factors. Therapy, if it is forthcoming, should be directed initially at these two key phenomena.

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